The herpes simplex virus (HSV 1) generates viral particles and infects mucus membrane cells before moving onto sensory nerve cells where they make their way to the brain. This intracellular transportation system can now be observed through the joint efforts of the three research bodies, and they delve deeper into its relationship with cognitive decline and Alzheimer’s disease.
The relationship between cellular APP and viral particles has been found to cause modifications in the cellular structure, of the cells affected by the herpes virus. APP is the largest constituent of senile plaques that are present in people with Alzheimer’s disease.
The results released showed that the majority of intracellular HSV 1 particles go through an interplay with APP and cause disruptions with its transportation and distribution process. It is widely accepted that HSV1 infections can lead to the development of Alzheimer’s disease, but the scientists are still trying to work out how significant its role is and how it interplays with other risk factors for cognitive decline.
While there are eight strains of the herpes virus that are known to infect people in various ways, doctors are still in the process of understanding how this elusive virus works. Firstly, it cannot be worked out of the body, and secondly, it enters a state of latency that has researchers unsure how it manages to evade the body’s immune system, resulting in untimely reactivations and infections that can take weeks to heal for some people. And, while antiviral medication has been proven to be effective when administered early on in an infection, the new study has not yet isolated whether treatment for HSV 1 can lower a person’s chances of developing Alzheimer’s.
Despite the progress, early treatment still provides the best prognosis for recovery, and low cost clinics in Las Vegas provide affordable treatment to help you recover quicker.